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M on May 12th, 2012

After what seems like forever waiting to find one, we went and picked up our new house cow today.  She is purebred Jersey, will be four years old in October and has a five month old steer calf at foot – he has tentatively been named “Bangers” as in ‘bangers and mash’ to go with […]

Continue reading about We have a Jersey milk cow!

M on September 30th, 2011

Little T-Bone is learning that its ok to spend some time away from his mother and that when he is tied up he has to stay in that spot!  We are progressing fairly well with the halter and lead training…the catching him in the paddock however requires considerable work.  He might look cute, but in […]

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M on September 7th, 2011

After many weeks of looking like she was going to pop any moment, Charlotte finally had her calf this morning just after 10am.  She had a fairly quick and easy birth and we were lucky enough to be able to watch, photograph and video the whole birth which was pretty exciting seeing as it was […]

Continue reading about Our firstborn calf

Serenade with satirical whistle

Older men with diabetes mellitus, but also severely overweight men, may develop secondary hypogonadism. "The core symptom of low testosterone levels is usually decreased libido," Dr. Cornelia Jaursch-Hancke from the German Clinic for Diagnostics, Sydney, at the conference in Melbourne. Various additional symptoms such as osteoporosis, anemia, erectile dysfunction, decreasing muscle strength and mass, but also diminishing vitality and depression can be added. In secondary hypogonadism the function of the hypothalamus or pituitary is impaired so that the Leydig cells of the testicle no longer form testosterone or no testosterone due to lack of stimulation. Typically, the gonadotropins LH and FSH in the serum are still normal to low. This also applies to patients with type 2 diabetes, of which about 25 to 50 percent are affected, the endocrinologist reported. An increasing problem is also MOSH, the "male obesity associated secondary hypogonadism". As a cause, she described the visceral fetal cells, which are highly active endocrinically and produce mediators, which promote, inter alia, insulin resistance, inflammatory processes and dyslipidemia and stimulate estradiol production. In sum, the hypothalamic-pituitary axis is inhibited. Continue reading...

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