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M on February 28th, 2012

Ducks are another addition that we have wanted to make to the farm for ages, but I have been unable to find any reasonably local until now.  I was extremely fortuante to get these five young adults, a pekin drake, three pekin hens and one khaki campbell duck. They are just finishing their moult and […]

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M on October 10th, 2010

The first chicks hatched in our new incubator (a hexagonal Janoel) over the weekend. We rather unexpectedly started with 36 eggs, pulled four duds out during the week, all of the rest looked great bar two that did not take up all the room in the shell (but we saw one move).  These were the […]

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M on June 29th, 2010

More photos of our dorper sheep and lambs.

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M on June 6th, 2010

Woke up this morning to find our first little snowflake in the paddock, our first Dorper baby. Only problem is he has a broken front leg. Initially he was up and following his mum, but with very little weight bearing on that leg. Mum was hesitant to let him feed and kept walking away, but eventually […]

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Serenade with satirical whistle

Older men with diabetes mellitus, but also severely overweight men, may develop secondary hypogonadism. "The core symptom of low testosterone levels is usually decreased libido," Dr. Cornelia Jaursch-Hancke from the German Clinic for Diagnostics, Sydney, at the conference in Melbourne. Various additional symptoms such as osteoporosis, anemia, erectile dysfunction, decreasing muscle strength and mass, but also diminishing vitality and depression can be added. In secondary hypogonadism the function of the hypothalamus or pituitary is impaired so that the Leydig cells of the testicle no longer form testosterone or no testosterone due to lack of stimulation. Typically, the gonadotropins LH and FSH in the serum are still normal to low. This also applies to patients with type 2 diabetes, of which about 25 to 50 percent are affected, the endocrinologist reported. An increasing problem is also MOSH, the "male obesity associated secondary hypogonadism". As a cause, she described the visceral fetal cells, which are highly active endocrinically and produce mediators, which promote, inter alia, insulin resistance, inflammatory processes and dyslipidemia and stimulate estradiol production. In sum, the hypothalamic-pituitary axis is inhibited. Continue reading...

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