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After many weeks of looking like she was going to pop any moment, Charlotte finally had her calf this morning just after 10am.  She had a fairly quick and easy birth and we were lucky enough to be able to watch, photograph and video the whole birth which was pretty exciting seeing as it was our first calf.  I admit that I was a little concerned that I would have to intervene as Charlotte is nowhere near as tamed down as Matilda, so it was a relief that everything went like clockwork.

We were finally able to determine that the calf is a bull and because he will be going to Freezer Camp when he is older, his name is T-Bone.  We were hoping that the heifer fairy would visit so that we could keep rather than eat our first calf, but the main thing is that mum and baby are doing fine…and the hand milking can begin shortly!

Charlotte- Early labour

Calf's feet appear in the sac

Charlotte down and calf coming out with Midwife Matilda standing by

Charlotte breaking the sac at the calf's mouth

Trying to stand

Faceplant!

Lean on Me

Finally stable on his legs

Baby T-Bone

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Older men with diabetes mellitus, but also severely overweight men, may develop secondary hypogonadism. "The core symptom of low testosterone levels is usually decreased libido," Dr. Cornelia Jaursch-Hancke from the German Clinic for Diagnostics, Sydney, at the conference in Melbourne. Various additional symptoms such as osteoporosis, anemia, erectile dysfunction, decreasing muscle strength and mass, but also diminishing vitality and depression can be added. In secondary hypogonadism the function of the hypothalamus or pituitary is impaired so that the Leydig cells of the testicle no longer form testosterone or no testosterone due to lack of stimulation. Typically, the gonadotropins LH and FSH in the serum are still normal to low. This also applies to patients with type 2 diabetes, of which about 25 to 50 percent are affected, the endocrinologist reported. An increasing problem is also MOSH, the "male obesity associated secondary hypogonadism". As a cause, she described the visceral fetal cells, which are highly active endocrinically and produce mediators, which promote, inter alia, insulin resistance, inflammatory processes and dyslipidemia and stimulate estradiol production. In sum, the hypothalamic-pituitary axis is inhibited. Continue reading...

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