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We ended up with 25 hatched chicks, the remaining seven chicks died in the shell without pipping (2 of which I believed died around day 16).

Of the 25, one had *sort of* splayed legs – I’m not convinced they were splayed at least not like the photos I have seen on splayed legs – he also had a soft abdomen and was one of the ones stuck in the shell on day 23 – this little one died over night:( Another six have  slipped tendon (Perosis or “chick hock”) on one leg – as all six of these chicks were obtained from the same place, I believe it is a genetic or vitamin deficiency in the breeding stock.

I have tried taping and splinting the legs after repositioning the tendon, made slings in a small box to support their weight off the injured leg to give them a chance to heal, as well as put them on vitamins and minerals. Initially they were all managing to hop on one leg and eat and drink and looked healthy and alert, however over several days it became obvious that they were not going to recover and to prevent any further pain they were humanely pts.  It was a very sad way to learn the importance of the health of breeding stock that I source my fertile eggs from.

The remaining 18 chicks are happy, healthy and doing really well.

Baby chickens © South West Photography

Baby chickens

Baby chickens © South West Photography

Baby chickens

Baby chickens © South West Photography

Baby chickens

Chick with slipped hock © South West Photography

Chick with slipped hock - arrow shows where the skin had broken at the joint

Chick with slipped hock

Another Chick with slipped hock

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Older men with diabetes mellitus, but also severely overweight men, may develop secondary hypogonadism. "The core symptom of low testosterone levels is usually decreased libido," Dr. Cornelia Jaursch-Hancke from the German Clinic for Diagnostics, Sydney, at the conference in Melbourne. Various additional symptoms such as osteoporosis, anemia, erectile dysfunction, decreasing muscle strength and mass, but also diminishing vitality and depression can be added. In secondary hypogonadism the function of the hypothalamus or pituitary is impaired so that the Leydig cells of the testicle no longer form testosterone or no testosterone due to lack of stimulation. Typically, the gonadotropins LH and FSH in the serum are still normal to low. This also applies to patients with type 2 diabetes, of which about 25 to 50 percent are affected, the endocrinologist reported. An increasing problem is also MOSH, the "male obesity associated secondary hypogonadism". As a cause, she described the visceral fetal cells, which are highly active endocrinically and produce mediators, which promote, inter alia, insulin resistance, inflammatory processes and dyslipidemia and stimulate estradiol production. In sum, the hypothalamic-pituitary axis is inhibited. Continue reading...

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